PF-Atlas › Mechanisms › mTOR inhibition reduces cellular senescence, autophagy dysfunction, and fibroblast proliferation in lung
mTOR inhibition reduces cellular senescence, autophagy dysfunction, and fibroblast proliferation in lung in IPF
Mechanism of action · idiopathic pulmonary fibrosis
Overview
mTOR inhibition reduces cellular senescence, autophagy dysfunction, and fibroblast proliferation in lung is a therapeutic target in idiopathic pulmonary fibrosis. PF-Atlas tracks 1 drug and 0 clinical trials acting through this mechanism.
Drugs targeting mTOR inhibition reduces cellular senescence, autophagy dysfunction, and fibroblast proliferation in lung
Therapies
Medical disclaimer. This is a research summary, not medical advice.